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Using mammalian cells in culture, we found that micromolar levels of homocysteine decreased GPx1 expression and activity without changing the levels of GPx1 mRNA [68]

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Oxidative modifications to proteins, lipids, and/or DNA can alter the function of these molecules and may result in the activation of ER stress, mitochondrial dysfunction, autophagy, and apoptosis, processes that themselves also generate ROS [14]