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These effects are mediated through downstream activation of transcription factors such as c-Fos, c-Jun, and EGR-1, which regulate genes involved in cell cycle progression, extracellular matrix remodeling, and angiogenic signaling [26]

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Moreover, CCL5 (known as RANTES) plays a significant role in the establishment and maintenance of the inflammatory microenvironment in vitiligo by chemotactically attracting monocytes and macrophages via its receptor CCR5 (105, 106)