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It was discovered by sequencing the follicular GCs of OEM patients and normal female follicular GCs that the differently expressed genes were primarily abundant in the MAPK, Protein WNT(WNT) signaling pathway, apoptosis, and steroid hormone response (54)

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Interestingly, GSTM1 can also mediate similar inhibitory effects, but unlike GSTP1, GSTM1 is able to directly bind and inhibit the activity of ASK1 [67]